d. The psychosocial work environment
e. The demand-control model
f. The effort-reward imbalance model
g. Conclusion
h. References
Introduction
There has been a slow recognition that the importance of work for health goes beyond
traditional occupational diseases (Schilling1989). Indeed, it is likely that work makes a
greater contribution to diseases and ill-health not thought of as 'occupational.
Early research concentrated on the possible role of physical activity in the work place
(Morris et al, 1953). Other work, more in the spirit of traditional occupational health,
has specified a number of physical and chemical exposures (e.g. lead, carbon disulphide,
carbon monoxide, nitroglycerin, nitroglycol (Kristensen1994)). More recently the workplace
has been seen as an appropriate setting for health promotion activities: providing the
opportunity to influence lifestyles such as smoking, diet and physical activity, and to
conduct screening for disease risk (Breucker and Schroer 1996).
There is now evidence that psychosocial factors at work may play an important role in
contributing to the social gradient in ill health. There have been a number of different
approaches to measurement of work stress, and research more recently has tended to focus
on a few explicit theoretical concepts. Among these, the models of job demand-control
(Karasek1979, Karasek and Theorell 1990) and effort-reward imbalance (Siegrist et al,
1986, Siegrist 1996) have received special attention.
A number of different diseases have been related to psychosocial conditions in the
workplace, most notably coronary heart disease (CHD), musculoskeletal disorders and mental
illness. This paper touches on two types of question: the relation between conditions at
work and disease; and the contribution this relationship may make to variations in disease
in society. Because variations in coronary heart disease have been studied extensively, we
start and end with that disease, but as the paper will endeavour to show a number of other
disease end-points are important.
The Social Distribution of Coronary Heart Disease
It is worth reviewing the changing distribution of CHD internationally to put into
context the possible contribution of work stress to the development of ill health and
disease. There have been two major changes in the epidemiology of CHD over recent years:
i) a changing social class distribution of the disease (Marmot1992) and (ii) a rise and
fall in CHD in different countries (Uemura and Pisa1988). In many European countries, as
in the USA, as CHD became a mass disease, it rose first in higher socio-economic groups
and subsequently in lower, to the extent that the social distribution changed to the now
familiar pattern of an inverse social gradient: higher rates as the social hierarchy is
descended. More recently, the decline in CHD mortality both in the United Kingdom and the
USA has been enjoyed to a greater extent by higher socio-economic groups leading to a
widening of the social gap (Wing et al, 1992). Concerns that the predominance of CHD in
higher socio-economic groups may relate to the stress of their occupations go back at
least to Osler (1910) who wrote that work and worry were major causes of the disease. The
fact that CHD is now more common in lower socio-economic groups does not, by itself,
refute the potential importance of work 'stress'. Research has moved on from the
simplistic notion that high responsibility or dealing with multiple tasks represents work
stress.
There is now a widely validated body of knowledge on risk factors for CHD that relate
to development of atherosclerosis, and a somewhat less secure body of knowledge relating
to predisposition to thrombosis. The major risk factors are high levels of blood pressure
and plasma total cholesterol and smoking. Although smoking, in particular, shows a strong
social gradient (Marmot et al, 1991), these risk factors account for no more than one
third of the social gradient in cardiovascular disease (Marmot et al, 1978, Marmot et al,
1984).
We are left then with two types of question. First, what accounts for the social and
international variation in unhealthy behaviours such as atherogenic diet, smoking, and
sedentary life style? Second, given that these factors appear to be inadequate
explanations of social and international variations in cardiovascular mortality, what else
could account for the observed differences? We have argued elsewhere that one must look
for explanations in the nature of social and economic organisation of societies (Marmot
1994). One particular feature is the nature of working life, both because what happens in
the work place may be important for health and because work and the operation of the
labour market play a central role in the organisation of social and economic life, which
in turn are important in the social determinants of health. The evidence that supports the
importance of work for cardiovascular and other diseases is presented below.
The Changing Nature of Work
There are at least four important reasons for the centrality of work and occupation in
advanced industrialized societies. First, having a job is a principal pre-requisite for
continuous income opportunities. Level of income determines a wide range of life chances.
Second, training for a job and achievement of occupational status are the most important
goals of primary and secondary socialization. It is through education, job training, and
status acquisition that personal growth and development are realized, that a core social
identity outside the family is acquired, and that intentional, goal-directed activity in
human life is shaped. Third, occupation defines a most important criterion of social
stratification in advanced societies. Amount of esteem and social approval in
interpersonal life largely depend on the type of job, professional training, and level of
occupational achievement. Furthermore, type and quality of occupation, and especially the
degree of self-direction at work, strongly influence personal attitudes and behavioral
patterns in areas that are not directly related to work, such as leisure, family life,
education, and political activity (Kohn and Schooler 1973). Finally, occupational settings
produce the most pervasive and continuous demands during one´s lifetime, and they absorb
the largest amount of active time in adult life. Exposure to adverse job conditions
carries the risk of ill health by virtue of the amount of time spent and the quality of
demands faced at the workplace. At the same time, occupational settings provide unique
opportunities to experience reward, esteem, success, and satisfaction. To understand the
impact of working life on health in general, it is important to realize the profound
changes that have taken place in the nature of work in established market economies. Among
these are the following:
- fewer jobs are defined by physical demands; more by psychological and emotional demands;
- fewer jobs are available in mass production, more in the service sector;
- more jobs are concerned with information processing due to computerization and
automation.
These changes in the nature of work have gone along with changes in the nature of the
labour market. There has been increasing participation of women in the labour market, an
increase in short term and part-time working and, most importantly, an increase in job
instability and structural unemployment. For instance, Hutton (1995) describes Britain as
the 40-30-30 society: 40% of the male population of working age have secure jobs, 30% are
not working, and 30% are in insecure jobs. The 30% not working may cause some surprise
given that the official unemployment rate is around 8%. The 30% is made up of the official
unemployed, those no longer seeking work, premature retirements, disabled and others. If
30% of the population are in insecure jobs, this must have effects on the rest of the
working population who wonder if their job is next. This is a change. In Europe, until
relatively recently there were national commitments to security of employment. Now, the
rhetoric is labour market flexibility (Beatson1995). The other side of flexibility is job
insecurity.
The 30% not working is not unique to Britain. In Finland, for example, the mean age of
entry to the labour market is now 27 and mean age of exit is 53. When the Finnish social
contract was nationally agreed the assumption was that working life would last 40 years.
If it lasts 26 years, on average, this has a profound importance for the costs of the
welfare state. It also changes attitudes to work if a job for life is no longer a
realistic expectation for large sections of the labour market. Research on work and health
has to take this job insecurity into account, especially so as loss of job was shown to be
associated with elevated risk of mortality in independent prospective studies both in
Britain and in Finland (Morris et al, 1994, Martikainen and Valkonen 1996).
This changing nature of work and the labour market has occurred at the same time as
there have been substantial increases in income inequalities in many countries (Joseph
Rowntree Foundation 1995). Wilkinson (1992) has shown that, internationally, life
expectancy is related more closely to income distribution than to overall wealth as
measured by gross national product. This has now been documented in two independent
studies for the states of the USA (Kaplan et al, 1996, Kawachi and Kennedy 1997). If
inequality, rather than absolute level of deprivation is an important driver of health
differentials it may, as Wilkinson suggests in this book, be a reflection of the quality
of the social environment. It may also suggest that discontent related to unfavorable
social comparison (relative social deprivation) and associated stress reactions may have
important health consequences.
The scientific challenge, then, consists in identifying those stress-eliciting
conditions related to the nature of work, the structure of salaries (income distribution)
and labour market constraints that may account for differences in morbidity and mortality
that are reported within and between populations.
The Psychosocial Work Environment
Research on psychosocial work-related stress differs from traditional biomedical
occupational health research by the fact that stressors cannot be identified by direct
physical or chemical measurements. Rather, theoretical concepts are needed to analyse the
nature of work in order to identify particular stressful job characteristics at a level of
generalization that allows for their identification in a wide range of different
occupations.
These theoretical concepts are operationalized using standardized methods of social and
behavioral sciences (e.g. systematic observation, structured interviews, standardised
questionnaires (so called paper and pencil tests)). Therefore, measuring stressful working
conditions provides a theoretical and methodological challenge. As mentioned, in
theoretical terms, those components of working life need to be identified that produce
intense, recurrent, and long lasting stressful experience at least in a substantial
proportion of those exposed. Moreover, researchers have to argue whether they restrict
their formulations to particular job characteristics or whether they analyze stressful
work experience in terms of an interaction of work characteristics and of coping
characteristics of the working person.
At a methodological level, measures of work stress are expected to be reliable,
sensitive to change, and valid. Two theoretical models: the demand-control model and
effort-reward imbalance model fulfill these methodological criteria and identify stressful
working conditions that are widely prevalent in advanced marked economies, such as changes
in task profiles, work control, structure of salaries, and occupational stability. Over
the past ten years these two models have been tested in a number of studies, and a
substantial body of knowledge has been generated, strengthening the assumption that
stressful experiences at work are associated with elevated risk of CHD and other diseases.
The Demand-Control Model 
In the sixties, research on job conditions and CHD had explored working demands and
working hours (Hinkle et al, 1968). In the seventies several research traditions found
evidence for a favorable effect on mental health produced by skill development (Hackman
and Lawler1971) and autonomy at work (Kohn and Schooler 1973, Gardell 1971). It was
Karasek's original contribution to formulate a two-dimensional concept of work stress
where a high level of psychological demands combined with a low level of decision latitude
(low level of decision authority and low level of skill utilization) was predicted to
increase the risk of stressful experience and subsequent physical illness (in particular
CHD) (Karasek1979). In 1981, Karasek first found evidence of a predictive role of high
demand-low control conditions in CHD, using data on a representative Swedish sample
(Karasek et al, 1981). Since then, a large number of prospective and cross-sectional
studies on associations of stressful work as defined by high demand and low control (job
strain) with cardiovascular risk and disease have been conducted (for overviews see
(Karasek and Theorell 1990, Schnall and Landsbergis 1994, Kristensen 1995, Theorell and
Karasek 1996, Hemingway and Marmot 1998). A number of these studies have focused on
methodological considerations and have used new outcome measures, the majority of which
have revealed positive findings.
Karasek's original hypothesis that excessive psychological demands interact with lack
of decision latitude in generating increased risk of cardiovascular disease was
supplemented by a second hypothesis which concerns the learning of new patterns of
behaviour and skills on the basis of psychosocial job experience. According to this,
learning for adults accrues over a lifetime of work experience. It may contribute to the
worker's possibility to exert control over his or her working situation and thus have an
impact on broader conditions of adult life. According to this hypothesis, the active
situation is associated with the development of a feeling of mastery which inhibits the
perception of strain during periods of overload, for instance. This makes it likely that
the active job situation may stimulate healthy functioning. Epidemiological studies in
Sweden indicated that the active job situation is associated with high rates of
participation in socially active leisure and political activities (see Karasek and
Theorell 1990), and, on the contrary, the daily residual strain arising in the strain
situation gives rise to accumulated feelings of frustration which may inhibit learning
attempts. It is obvious that some of the 'classic' high strain jobs are found in mass
industry, especially under conditions of piece work and machine paced assembly line work.
Nevertheless, a number of strain jobs were also identified in the service sector. The
concept therefore proves to be relevant in different employment sectors, and will remain
important in the foreseeable future due to changing patterns of employment. For example,
the rate of temporary employment is increasing in western Europe, particularly for those
with low education. It is in these kinds of employment that lack of control will be a
major problem. Even in those with a high education, the increasing demands for flexibility
will create new decision latitude problems. The ever increasing demands for effectiveness
from the workforce are raising the levels of psychological demands for all workers. This
is particularly reflected in Swedish national welfare statistics.
More recently, the original demand-control concept was modified to include social
support at work as a third dimension (Johnson and Hall 1988) and to assess work control in
a life-course perspective 'total job control exposure' (Johnson et al, 1990). Another
important innovation concerns the exploration of health effects produced by intervention
studies that are based on the theoretical concept, and several promising intervention
studies have been reported recently (Theorell1992, Orth-Gomer et al, 1994, Karasek 1992).
Conclusion
In this paper we have argued that two theoretical models hold particular promise in
explaining at least part of the variation in CHD - a variation that may be attributed in
part to work stress as defined by the demand-control and effort-reward imbalance models.
High demand-low control conditions and high cost-low gain conditions at work are unequally
distributed both between and within societies and may potentially provide a framework in
which to understand the contribution of psychosocial factors at work to the development of
disease.
The conceptual differences between the models have direct implications for the design
of intervention measures to improve health; whereas the emphasis of the demand-control
model is on change of the task structure (such as job enlargement, job enrichment and
increasing the amount of support within the job etc.) the reduction of high cost-low gain
conditions includes action at three levels, the individual level (e.g. reduction of
excessive need for control), the interpersonal level (e.g. improvement of esteem reward),
and the structural level (e.g. adequate compensation for stressful work conditions by
improved pay and related incentives, opportunities for job training, learning new skills
and increased job security).
Despite the central role of work in the above models, an exclusive focus on working
life runs the risk of underestimating the true costs on health produced by other adverse
stressful circumstances that can occur outside of work. This becomes dramatically clear if
we consider the evidence on the health burden of long-term unemployment (Martikainen and
Valkonen 1996). The characteristics of family life and leisure activities are also of
crucial importance in reducing the stresses and strains of working life. Conversely,
stressful events in an individuals personal life, such as marital problems and lack
of social support can also exacerbate the burden of work-related stress and may increase a
person's disposition towards developing disease. The study of the work-family interface
points to the need to extend the framework of reference in stress research by taking into
account the broader social determinants of health.
References
Aust, B. Peter, R. and Siegrist, J. (1997). Stress management in bus drivers: a pilot
study based on the model of effort-reward imbalance. International Journal of Stress
Management, 4, 297-305.
Beatson, M. (1995). Labour market flexibility, London: Dept of Employment.
Bosma, H. Marmot, M.G. Hemingway, H. Nicholson, A. Brunner, E.J. and Stansfeld, S.
(1997). Low job control and risk of coronary heart disease in the Whitehall II
(prospective cohort) study. British Medical Journal, 314, 558-565.
Bosma, H. Peter, R. Siegrist, J. and Marmot, M.G. (1998). Alternative job stress models
and the risk of coronary heart disease. American Journal of Public Health, 88,
68-74.
Breucker, G.S. and Schroer, A. (eds.) (1996). International experiences in workplace
health promotion, European Health Promotion. Series 6, Copenhagen: WHO.
Gardell, B. (1971). Alienation and mental health in the modern industrial environment.
In: Levi, L. (ed.) Society, stress and disease. The psychosocial environment and
psychomatic diseases, pp. 148-180. London: Oxford University Press.
Greiner, B.A. Ragland, D.R. Krause, N. Syme, S.L. Fisher, J.M. (1997). Objective
measurement of occupational stress factors - an example with San Francisco urban transit
operators. Journal of Occupational Health Psychology, 4, 325-342.
Hackman, J.R. and Lawler, E.E. (1971). Employee reactions to job characteristics. Journal
of Applied Psychology, 55, 259-286.
Hallqvist, J. Diderichsen, F. Theorell, T. Reuterwall, C. Ahlbon, A. and the SHEEP
study (1998). Is the effect of job strain on myocardial infarction due to interaction
between high psychlogical demands and low decision latitude. Results from the Stockholm
Heart Epidemiology Program (SHEEP). Social Science and Medicine 46
(11):1405-1415.
Hemingway, H. Shipley, M. Stansfeld, S. and Marmot, M. (1997). Back pain sickness
absence, psychosocial work characteristics and employment grade: a prospective study in
office workers. Scandinavian Journal of Work and Environmental Health, 23,
121-129.
Hemingway, H. and Marmot, M. (1998). Psychosocial factors in the primary and secondary
prevention of coronary heart disease: a systematic review. In: Yusuf, S. Cairns, J. Camm,
J. Fallen, E. and Gersch, B. (eds.) Evidence based cardiology, pp 269-285. London:
BMJ Publishing Group.
Hinkle, L.E. Whitney, L.H. Lehman, E.W. Dunn, J. Benjamin, B. King, R. Plakun, A. and
Flehinger, B. (1968). Occupation, education and coronary heart disease. Science 161,
238-246.
Hlatky, M.A. Lam, L.C. Lee, K.L. Clapp-Channing, N.E. Williams, R.B. Pryor, D.B.
Califf, R.M. and Mark, D.B. (1995). Job strain and the prevalence and outcome of coronary
artery disease. Circulation, 92, 327-333.
Hutton, W. (1995) High risk. Guardian, 2-3.
Johnson, J.V. and Hall, E.M. (1988). Job strain, work place social support, and
cardiovascular disease: a cross-sectional study of a random sample of the Swedish working
population. American Journal of Public Health, 78, 1336-1342.
Johnson, J.V. Stewart, W. Fredlund, P. (1990). Psychosocial Job Exposure Matrix: An
Occupationally Aggregated Attribution System for Work Environment Exposure Characteristics,
221th edn. Stockholm: National Institute for Psychosocial Factors and Health.
Joksimovic, L. Siegrist, J. Peter, R. Meyer-Hammar, M., Klimek, W. and Heintzen, M.
(1998). Psychosocial factors and restonosis after PTCA: the role of work-related
overcommitment. Submitted.
Joseph Rowntree Foundation (1995). Inquiry into income and wealth chaired by Sir
Peter Barclay, York: Joseph Rowntree Foundation.
Kaplan, G.A. Pamuk, E.R. Lynch, J.W. Cohen, R.D. and Balfour, J.L. (1996). Inequality
in income and mortality in the United States: analysis of mortality and potential
pathways. British Medical Journal, 312, 999-1003.
Karasek, R.A. (1979). Job demands, job decision latitude and mental strain:
implications for job design. Administrative Science Quarterley, 24, 285-308.
Karasek, R. Baker, D. Marxer, F. Ahlbom, A. and Theorell, T. (1981). Job decision
latitude, job demands and cardiovascular disease: a prospective study of Swedish men. American
Journal of Public Health, 71, 694-705.
Karasek, R. and Theorell, T. (1990). Healthy work: stress, productivity, and the
reconstruction of working life. New York: Basic Books.
Karasek, R. (1992). Stress prevention through work reorganisation: a summary of 19
intervention studies. Conditions of work digest, 11 pp. 23-41. Switzerland:
International Labour Office.
Kawachi, I. and Kennedy, B.P. (1997). Health and social cohesion: why care about income
inequality? British Medical Journal, 314, 1037-1040.
Kohn, M. and Schooler, C. (1973). Occupational experience and psychological
functioning: An assessment of reciprocal effects. American Sociological Review, 38
97-118.
Kristensen, T.S. (1994). Cardiovascular diseases and work environment. In:
Cherermisinoff, P.N. (ed.) Encyclopedia of environmental control technology, pp.
217-43. Houston: Gulf Publishing Company.
Kristensen, T.S. (1995). The demand-control-support model: Methodological challenges
for future research. Stress Medicine 11, 17-26.
Marmot, M.G. Adelstein, A.M. Robinson, N. and Rose, G. (1978). The changing social
class distribution of heart disease. British Medical Journal, 2, 1109-1112.
Marmot, M.G. Shipley, M.J. and Rose, G. (1984). Inequalities in death - specific
explanations of a general pattern. Lancet i, 1003-6.
Marmot, M.G. Davey Smith, G. Stansfeld, S.A. Patel, C. North, F. Head, J. White, I.
Brunner, E.J. and Feeney, A. (1991). Health inequalities among British Civil Servants: the
Whitehall II study. Lancet, 337, 1387-1393.
Marmot, M.G. (1992). Coronary heart disease: rise and fall of a modern epidemic. In:
Marmot, M.G. and Elliott, P. (eds.) Coronary Heart Disease Epidemiology, pp. 3-19.
Oxford: Oxford University Press.
Marmot, M.G. (1994). Social differentials in health within and between populations. Daedalus,
123, 197-216.
Marmot, M.G. and Feeney, A. (1996). Work and health: implications for individuals and
society. In: Blane, D. Brunner, E. Wilkinson R. (eds.) Health and Social Organisation,
pp 235-54. London, New York: Routledge.
Martikainen, P.T. and Valkonen, T. (1996). Excess mortality of unemployed men and women
during a period of rapidly increasing unemployment. Lancet, 348, 909-912.
Morris, J.N. Heady, J.A. Raffle, P.A.B. Roberts, C.G. and Parks, J.W. (1953). Coronary
heart disease and physical activity of work. Lancet, 1053-1057.
Morris, J.K. Cook, D.G. and Shaper, A.G. (1994). Loss of employment and mortality. British
Medical Journal, 308, 1135-1139.
Netterstrom, B. and Suadicani, P. (1993). Self-assessed job satisfaction and ischaemic
heart disease mortality: a 10 year follow up of urban bus drivers. International
Journal of Epidemiology, 22, 51-56.
North, F. Syme, S.L. Feeney, A. Head, J. Shipley, M.J. and Marmot, M.G. (1993).
Explaining socioeconomic differences in sickness absence: the Whitehall II study. British
Medical Journal, 306, 361-366.
North, F.M. Syme, S.L. Feeney, A. Shipley, M. and Marmot, M. (1996). Psychosocial work
environment and sickness absence among British civil servants: The Whitehall II Study. American
Journal of Public Health, 86, 332-340.
Orth-Gomer, K. Eriksson, I. Moser, V. Theorell, T. and Fredlund, P. (1994). Lipid
lowering through work stress reduction. International Journal of Behavioural Medicine,
3, 204-214.
Osler, W. (1910). The Lumleian Lectures on angina pectoris. Lancet, i,
839-844.
Peter, R. Alfredsson, L. Hammar, N. Siegrist, J. Theorell, T. and Westerholm, P.
(1998a). High effort, low reward and cardiovascular risk factors in employed Swedish men
and women - baseline results from the WOLF study. Journal of Epidemiology and Community
Health, 52, 540-547.
Peter, R. Geissler, H. and Siegrist, J. (1998b). Associations of effort-reward
imbalance at work and reported symptoms in different groups of male and female public
transport workers. Stress Medicine, 14, 175-182.
Peter, R. and Siegrist, J. (1998). Chronic work stress, sickness absence and
hypertension in middle managers: general or specific sociological explanations. Social
Science and Medicine, 45, 1111-1120.
Reed, D.M. Lacroix, A.Z. Karasek, R.A. Miller, D. and MacLean, C.A. (1989).
Occupational strain and the incidence of coronary heart disease. American Journal of
Epidemiology, 129, 495-502.
Schilling, R.S.F. (1989). Health protection and promotion at work. British Journal
of Industrial Medicine, 46, 683-688.
Schnall, P.L. and Landsbergis, P.A. (1994). Job strain and cardiovascular disease. Annual
Review of Public Health, 15, 381-411.
Siegrist, J. Siegrist, K. Weber, I. (1986). Sociological concepts in the etiology of
chronic disease: the case of ischaemic heart disease. Social Science and Medicine, 22,
247-53.
Siegrist, J. Peter, R. Junge, A. Cremer, P. and Seidel, D. (1990). Low status control,
high effort at work and ischemic heart disease: prospective evidence from blue-collar men.
Social Science and Medicine, 31, 1127-1134.
Siegrist, J. (1996).Adverse health effects of high-effort/low-reward conditions. Journal
of Occupational Health Psychology, 1, 27-41.
Siegrist, J. Peter, R. Cremer, P. Seidel, D. (1997). Chronic work stress is associated
with atherogenic lipids and elevated fibrinogen in middle-aged men. Journal of Internal
Medicine, 242, 149-56.
Stansfeld, S.A. Fuhrer, R. Head, J. Ferrie, J. and Shipley, M. (1997). Work and
psychiatric disorder in the Whitehall II study. Journal of Psychosomatic Research, 43,
73-81.
Stansfeld, S. Bosma, H. Hemingway, H. and Marmot, M. (1998). Psychosocial work
characteristics and social support as predictors of SF-36 functioning: the Whitehall II
study. Psychosomatic Medicine, 60, 247-255.
Stansfeld, S. Bosma, H. Hemingway, H. and Marmot, M. (1999) Work characteristics
predict psychiatric disorder: prospective results from the Whitehall II study. Occupation
and Environmental Medicine, 56(5), 302-307.
Suadicani, P. Hein, H.O. and Gynetelberg, F. (1993). Are social inequalities associated
with the risk ischaemic heart disease a result of psychosocial working conditions? Atherosclerosis,
101, 165-175.
Theorell, T. (1992). Health promotion in the workplace. In: Badura, B. Kickbusch, I.
(eds.) Health promotion research. Towards a new social epidemiology, pp. 251-66.
Copenhagen: WHO.
Theorell, T. and Karasek, R.A. (1996). Current issues relating to psychosocial job
strain and cardiovascular disease research. Journal of Occupation and Health
Psychology, 1, 9-26.
Theorell, T. Tsutsumi, T. Hallqvist, J, Reuterwall, C. Fredlund, P. Emlund, N.
Alfredsson, L. Hammar, N. Ahlbom, A. Johnson, J. and the SHEEP Study. (1998). Decision
latitude, job strain, and myocardial infarction: a study of working men in Stockholm. American
Journal of Public Health, 88, 382-88.
Uemura, K. and Pisa, Z. (1988). Trends in cardiovascular disease mortality in
industrialised countries since 1950. World Health Statistical Quarterly, 41,
155-178.
Wilkinson, R.G. (1992). Income distribution and life expectancy. British Medical
Journal, 304, 165-168.
Wing, S. Casper M, Riggan, W. (1992). Geographic and socioeconomic variation in the
onset of decline of coronary heart disease mortality in white woman. American Journal
of Public Health, 82, 204-209.